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Number of items: 6.

Capper, Rebecca, Britt-Compton, Bethan, Tankimanova, Maira, Rowson, Janet Maud, Letsolo, Boitelo, Man, Stephen Tzekwung, Haughton, Michele Fleur and Baird, Duncan Martin 2007. The nature of telomere fusion and a definition of the critical telomere length in human cells. Genes and Development 21 (19) , pp. 2495-2508. 10.1101/gad.439107

Davis, Terence, Baird, Duncan Martin, Haughton, Michele Fleur, Jones, Christopher John and Kipling, David Glyn 2005. Prevention of accelerated cell aging in Werner syndrome using a p38 mitogen-activated protein kinase inhibitor. The Journals of Gerontology. Series A: Biological Sciences and Medical Sciences 60 (11) , pp. 1386-93.

Skinner, Julia, Bounacer, Ali, Bond, Jane Ann, Haughton, Michele Fleur, deMicco, Catherine and Wynford-Thomas, David 2004. Opposing effects of mutant ras oncoprotein on human fibroblast and epithelial cell proliferation: implications for models of human tumorigenesis. Oncogene 23 (35) , pp. 5994-5999. 10.1038/sj.onc.1207798

Preto, Ana, Singhrao, Simarjit Kaur, Haughton, Michele Fleur, Kipling, David Glyn, Wynford-Thomas, David and Jones, Christopher John 2004. Telomere erosion triggers growth arrest but not cell death in human cancer cells retaining wild-type p53: implications for antitelomerase therapy. Oncogene 23 (23) , pp. 4136-4145. 10.1038/sj.onc.1207564

Davis, Terence, Singhrao, Simarjit Kaur, Wyllie, Fiona Sandra, Haughton, Michele Fleur, Smith, Paul James, Wiltshire, Marie, Wynford-Thomas, David, Jones, Christopher John, Faragher, Richard G. A. and Kipling, David Glyn 2003. Telomere-based proliferative lifespan barriers in Werner-syndrome fibroblasts involve both p53-dependent and p53-independent mechanisms. Journal of Cell Science (JCS) 116 (7) , pp. 1349-57. 10.1242/jcs.00331

Stephens, Philip, Cook, Helen, Hilton, Joanne, Jones, Christopher John, Haughton, Michele Fleur, Wyllie, Fiona Sandra, Skinner, Julia W., Harding, Keith Gordon, Kipling, David Glyn and Thomas, David William 2003. An analysis of replicative senescence in dermal fibroblasts derived from chronic leg wounds predicts that telomerase therapy would fail to reverse their disease-specific cellular and proteolytic phenotype. Experimental Cell Research 283 (1) , pp. 22-35. 10.1016/S0014-4827(02)00021-6

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